The Potential Protective Role of Ketones and Ketosis Against EMF-Induced Oxidative Stress
Meat Induced EMF Protection
By James Ibbotson An Kyle Davey
Introduction In recent years, there has been growing concern about the potential health effects of electromagnetic field (EMF) exposure. While the mechanisms are not fully understood, EMF has been associated with increased oxidative stress in various biological systems, including bacteria. Interestingly, recent research suggests that ketones, particularly beta-hydroxybutyrate (BHB), may have antioxidant properties that could help combat oxidative stress. This raises the intriguing possibility that being in a state of ketosis might offer protection against EMF-induced oxidative stress, not only for the host but also for the gut microbiome.
Ketones as Antioxidants Ketone bodies, especially BHB, have been shown to possess antioxidant properties. BHB can reduce oxidative stress through several mechanisms. First, it can increase the expression of antioxidant enzymes like superoxide dismutase and catalase, which help neutralize reactive oxygen species (ROS). Second, BHB can directly scavenge ROS, acting as a molecular "mop" to clean up these harmful molecules. Finally, BHB can enhance mitochondrial function and reduce mitochondrial ROS production, further reducing oxidative stress.
Beyond their antioxidant properties, ketones, particularly BHB, have interesting interactions with the blood-brain barrier (BBB). Ketones can cross the BBB more easily than glucose, allowing them to serve as an alternative energy source for the brain when glucose is scarce. Additionally, ketones may help maintain BBB integrity, reduce inflammation, and decrease oxidative stress in the brain. These properties make ketones a subject of interest in treating various neurological conditions.
Bacterial Utilization of Ketones It's not just the host that can benefit from ketones – certain gut bacteria have also been shown to metabolize these molecules. Some gut microbes can use ketones as an alternative energy source, while others may use them to regulate their redox balance. This suggests that ketones could potentially influence the metabolism and function of the gut microbiome, with implications for overall health.
EMF, Oxidative Stress, and Bacteria EMF exposure has been linked to increased oxidative stress in various biological systems, and bacteria are no exception. Studies have suggested that EMF can induce oxidative stress in bacteria, potentially influencing their growth and metabolism. This is particularly relevant given the growing understanding of the importance of the gut microbiome in human health.
A Hypothetical Protective Mechanism If bacteria can indeed use ketones to mitigate oxidative stress, they might be better equipped to handle EMF-induced ROS. In a state of ketosis, where ketone levels are elevated, the increased availability of these protective molecules could potentially benefit both the host and the gut microbiome. This could offer a multi-layered defense against EMF-induced oxidative stress: direct antioxidant effects in the host's cells, ketone utilization by gut bacteria to maintain redox balance, and potential changes in gut microbiome composition that favor more resilient or adaptive species.
An Analogy: The Tug-of-War Between Gram-Negative and Gram-Positive Bacteria To illustrate this hypothetical protective mechanism, consider the example of Klebsiella pneumoniae, a gram-negative bacterium that has shown a 25% increase in antibiotic resistance when exposed to Wi-Fi and 900 MHz frequencies. While K. pneumoniae may produce ketones to combat the EMF-induced oxidative stress, these same ketones could also provide a boost to gram-positive bacteria in the gut.
Imagine a tug-of-war between the gram-negative K. pneumoniae and the gram-positive bacteria. The ketones produced by K. pneumoniae in response to EMF stress may inadvertently give the gram-positive bacteria a "stronger rope," allowing them to pull harder and maintain their ground. This could explain why, despite the increased antibiotic resistance of K. pneumoniae in the presence of EMF, people don't often succumb to K. pneumoniae infections.
However, this "tug-of-war" comes at a cost. The increased production and utilization of ketones by both gram-negative and gram-positive bacteria may lead to a faster depletion of certain minerals, like magnesium, that are essential for maintaining the redox balance and overall health of the gut microbiome. This could contribute to the widespread magnesium deficiency seen in modern populations and highlight the importance of ensuring adequate magnesium intake, especially in the context of EMF exposure.
Research Implications and Future Directions To fully investigate this hypothesis, several lines of research are needed. First, studies should measure oxidative stress markers in gut bacteria exposed to EMF, both with and without the presence of ketones. This could help establish whether ketones can indeed protect bacteria from EMF-induced oxidative stress. Second, assessing changes in bacterial gene expression related to antioxidant defenses in response to ketones and EMF could provide insights into the molecular mechanisms at play. Finally, evaluating the impact of ketosis on gut microbiome composition and resilience to EMF-induced oxidative stress could reveal the potential for ketosis to promote a more robust and adaptive gut microbiome.
Furthermore, investigating the potential protective effects of ketones on the blood-brain barrier in the context of EMF exposure could be another promising avenue of research. Given that certain bacteria can invade the central nervous system by crossing the blood-brain barrier or the blood-cerebrospinal fluid barrier, understanding how ketones might influence these interactions could provide insights into protecting against bacterial meningitis in EMF-rich environments.
It's worth noting that the blood–central nervous system (CNS) barriers are tight and protect the brain parenchyma from insults, including those of infectious origin. This barrier function is due to the presence of tight junctions between the endothelial cells of the brain. A limited number of blood-borne bacteria can cross these barriers and cause meningitis, including Neisseria meningitidis, Streptococcus pneumoniae, and in newborns, group B Streptococcus and Escherichia coli K1. The level of bacteraemia plays a key role in meningeal tropism, and these extracellular bacteria interact directly with the blood–CNS barriers through various mechanisms.
Evolutionary Perspective: Bacterial Adaptation to EMF Stress Interestingly, the potential for bacteria to use ketones to handle EMF-induced oxidative stress may not be entirely new. It's possible that this mechanism could have evolved as a way for our gut microbes to cope with the increased EMF exposure that has occurred since the introduction of widespread radio communication and, in particular, since the influenza pandemic of 1918.
During this time, the world saw a rapid increase in the use of radio waves for communication, coinciding with one of the deadliest pandemics in modern history. It's conceivable that the gut bacteria of individuals who survived the 1918 pandemic may have adapted to the increased EMF stress by leveraging the protective effects of ketones, which would have been more readily available during the fasting and starvation states that were common during this period.
This evolutionary perspective adds another layer to the already compelling hypothesis that ketones and ketosis could offer protection against EMF-induced oxidative stress. It suggests that our gut microbes may have already developed strategies to cope with this type of stress, and that by entering a state of ketosis, we may be tapping into an ancient, evolutionary mechanism for promoting resilience and health.
Of course, this evolutionary hypothesis requires further investigation and validation. But it serves as a reminder of the incredible adaptability and resilience of our gut microbiome, and the potential for dietary interventions like ketosis to support and enhance these innate defense mechanisms.
Conclusion The idea that ketones could help defend against EMF-induced oxidative stress, both in the host and in the gut microbiome, is a fascinating and potentially groundbreaking hypothesis. It highlights the complex interplay between diet, the gut microbiome, and environmental stressors, and suggests new avenues for protecting health in an increasingly EMF-exposed world.
The potential protective role of ketones extends beyond just the gut microbiome and general oxidative stress. Their ability to interact with the blood-brain barrier and potentially offer neuroprotection adds another layer to their significance in the context of EMF exposure. As we continue to investigate the health implications of EMF exposure, the role of ketones in modulating these effects - from the gut to the brain - presents an exciting and promising area of focus.
While more research is needed to fully understand the mechanisms and implications of this hypothesis, the existing evidence supporting the antioxidant effects of ketones, the ability of bacteria to utilize these molecules, and the impact of EMF on bacterial oxidative stress provides a strong foundation for further exploration.
As we continue to investigate the health implications of EMF exposure and the role of diet and the gut microbiome in modulating these effects, the potential protective role of ketones and ketosis is an exciting and promising area of focus. This line of inquiry could have far-reaching implications for fields ranging from microbiology and environmental health to nutritional science and personalized medicine.
In a world where EMF exposure is increasingly ubiquitous, understanding and harnessing the protective potential of ketones and ketosis could be a powerful tool for promoting health and resilience. As research in this area advances, it may open up new strategies for mitigating the impact of EMF on both individual and public health, and could even inform the development of novel therapies and interventions.
Certainly. I'll draft a research paper suitable for peer review based on the hypotheses and ideas discussed in your blog post. This paper will follow a standard scientific format, including an abstract, introduction, methods, results, discussion, and conclusion sections. Please note that this is a hypothetical paper, as the specific experiments and results described have not actually been conducted. This draft is meant to serve as a framework for potential future research.
Title: Ketones as a Potential Protective Mechanism Against EMF-Induced Oxidative Stress in the Gut Microbiome
Abstract: Electromagnetic field (EMF) exposure has been associated with increased oxidative stress in various biological systems, including bacteria. This study investigates the hypothesis that ketones, particularly beta-hydroxybutyrate (BHB), may offer protection against EMF-induced oxidative stress in the gut microbiome. We examined the effects of BHB on oxidative stress markers in gut bacteria exposed to EMF, assessed changes in bacterial gene expression related to antioxidant defenses, and evaluated the impact of ketosis on gut microbiome composition and resilience to EMF-induced oxidative stress. Our findings suggest that BHB may indeed provide a protective effect against EMF-induced oxidative stress in gut bacteria, potentially through upregulation of antioxidant defense mechanisms and modulation of the gut microbiome composition. These results have implications for understanding the complex interplay between diet, the gut microbiome, and environmental stressors, and may inform strategies for mitigating the health impacts of EMF exposure.
Introduction: In recent years, concern has grown regarding the potential health effects of electromagnetic field (EMF) exposure. EMF has been associated with increased oxidative stress in various biological systems, including bacteria (Lai and Singh, 2004). Concurrently, research has highlighted the importance of the gut microbiome in human health and its potential susceptibility to environmental stressors (Clemente et al., 2012).
Ketone bodies, particularly beta-hydroxybutyrate (BHB), have been shown to possess antioxidant properties (Veech, 2014). BHB can reduce oxidative stress by increasing the expression of antioxidant enzymes, directly scavenging reactive oxygen species (ROS), and enhancing mitochondrial function (Newman and Verdin, 2014).
This study investigates the hypothesis that ketones may offer protection against EMF-induced oxidative stress in the gut microbiome. We propose that in a state of ketosis, where ketone levels are elevated, the increased availability of these protective molecules could benefit both the host and the gut microbiome, providing a multi-layered defense against EMF-induced oxidative stress.
Methods: Bacterial Cultures and EMF Exposure: We used a mixed culture of common gut bacteria, including Escherichia coli, Bacteroides fragilis, and Lactobacillus acidophilus. Cultures were exposed to EMF at 900 MHz (similar to cell phone frequencies) for 24 hours, with and without the presence of 2 mM BHB.
Oxidative Stress Markers: We measured levels of malondialdehyde (MDA) and 8-hydroxy-2'-deoxyguanosine (8-OHdG) as markers of lipid peroxidation and DNA oxidation, respectively.
Gene Expression Analysis: RT-qPCR was used to assess changes in expression of genes related to antioxidant defenses, including superoxide dismutase (SOD) and catalase (CAT).
Microbiome Composition Analysis: 16S rRNA gene sequencing was performed to evaluate changes in gut microbiome composition under different experimental conditions.
In Vivo Ketosis Model: We used a mouse model to induce ketosis through a ketogenic diet. Fecal samples were collected and analyzed for microbiome composition and oxidative stress markers following EMF exposure.
Results:
Oxidative Stress Markers: EMF exposure significantly increased levels of MDA and 8-OHdG in bacterial cultures (p < 0.001). The presence of BHB reduced these markers by 35% and 28%, respectively (p < 0.01).
Gene Expression: EMF exposure led to a 2.5-fold increase in SOD and CAT expression (p < 0.001). In the presence of BHB, this increase was further enhanced to 3.8-fold (p < 0.001).
Microbiome Composition: EMF exposure altered the relative abundance of certain bacterial species, with a 20% decrease in Lactobacillus spp. and a 15% increase in Enterobacteriaceae. BHB supplementation mitigated these changes, maintaining a composition more similar to the control group.
In Vivo Ketosis Model: Mice on a ketogenic diet showed increased resistance to EMF-induced changes in their gut microbiome composition and had lower levels of oxidative stress markers in fecal samples following EMF exposure compared to control diet mice (p < 0.01).
Discussion: Our results suggest that BHB may indeed provide a protective effect against EMF-induced oxidative stress in gut bacteria. The reduction in oxidative stress markers in the presence of BHB indicates its potential to mitigate EMF-induced damage. The enhanced expression of antioxidant genes in the presence of BHB suggests that ketones may boost the bacteria's innate defense mechanisms against oxidative stress.
The changes in microbiome composition observed with EMF exposure highlight the potential for environmental stressors to disrupt the gut ecosystem. The ability of BHB to maintain a more stable microbiome composition in the face of EMF exposure suggests that ketones may play a role in promoting microbiome resilience.
The in vivo results from our ketosis mouse model provide further support for the protective effect of ketones against EMF-induced changes in the gut microbiome. This aligns with our hypothesis that a state of ketosis could offer multi-layered protection against EMF-induced oxidative stress.
These findings have implications for understanding the complex interplay between diet, the gut microbiome, and environmental stressors. They suggest that dietary interventions, particularly those that promote ketosis, could potentially be used as a strategy to mitigate the effects of EMF exposure on gut health.
Conclusion: This study provides initial evidence supporting the hypothesis that ketones, particularly BHB, may offer protection against EMF-induced oxidative stress in the gut microbiome. Our findings suggest that this protection may be mediated through direct antioxidant effects, enhancement of bacterial antioxidant defenses, and promotion of a more resilient microbiome composition.
Future research should focus on elucidating the specific mechanisms by which ketones interact with different bacterial species to confer protection against EMF-induced stress. Additionally, long-term studies in humans are needed to determine the clinical relevance of these findings and to explore the potential of ketogenic diets or ketone supplementation as interventions to mitigate the health impacts of EMF exposure.
These results open up new avenues for research into the role of diet in modulating the effects of environmental stressors on human health, with potential implications for fields ranging from environmental health to personalized nutrition.
Title: Ketones as a Potential Protective Mechanism Against EMF-Induced Oxidative Stress: Implications for the Gut Microbiome and Beyond
Abstract: Electromagnetic field (EMF) exposure has been associated with increased oxidative stress in various biological systems, including bacteria. This study investigates the hypothesis that ketones, particularly beta-hydroxybutyrate (BHB), may offer protection against EMF-induced oxidative stress not only in the gut microbiome but potentially in the bloodstream as well. We examined the effects of BHB on oxidative stress markers in gut bacteria exposed to EMF, assessed changes in bacterial gene expression related to antioxidant defenses, and evaluated the impact of ketosis on gut microbiome composition and resilience to EMF-induced oxidative stress. Our findings suggest that BHB may indeed provide a protective effect against EMF-induced oxidative stress in gut bacteria and could potentially extend to the prevention of harmful blood cell aggregation. These results have broad implications for understanding the complex interplay between diet, the gut microbiome, and systemic health, and may inform strategies for mitigating the health impacts of EMF exposure.
Introduction: Electromagnetic field (EMF) exposure has been associated with increased oxidative stress in various biological systems, including bacteria (Lai and Singh, 2004). EMF-induced oxidative stress is thought to be mediated through the generation of reactive oxygen species (ROS), which can damage cellular components such as lipids, proteins, and DNA (Yakymenko et al., 2016). This oxidative stress has been linked to various adverse health outcomes, including cancer, neurodegenerative diseases, and immune system dysfunction (Kivrak et al., 2017).
Recent research has highlighted the importance of the gut microbiome in human health and its potential susceptibility to environmental stressors (Clemente et al., 2012). The gut microbiome plays a crucial role in many aspects of health, including nutrient metabolism, immune system regulation, and even mental health (Cryan and Dinan, 2012). Disruptions to the gut microbiome have been implicated in a wide range of diseases, from gastrointestinal disorders to metabolic and neurological conditions (Lynch and Pedersen, 2016).
Ketone bodies, particularly beta-hydroxybutyrate (BHB), have been shown to possess antioxidant and anti-inflammatory properties (Veech, 2014; Youm et al., 2015). BHB can reduce oxidative stress by increasing the expression of antioxidant enzymes, directly scavenging reactive oxygen species (ROS), and enhancing mitochondrial function (Newman and Verdin, 2014). These properties have led to interest in ketones as potential therapeutic agents for conditions associated with oxidative stress and inflammation, such as neurodegenerative diseases and cancer (Poff et al., 2015).
This study investigates the hypothesis that ketones may offer protection against EMF-induced oxidative stress not only in the gut microbiome but potentially in the bloodstream as well. We propose that in a state of ketosis, where ketone levels are elevated, the increased availability of these protective molecules could benefit the host, the gut microbiome, and the circulatory system, providing a multi-level defense against EMF-induced oxidative stress and its associated health risks. To our knowledge, this is the first study to examine the potential protective effects of ketones in the context of EMF exposure, the gut microbiome, and systemic health.
Methods: Bacterial Cultures and EMF Exposure: We used a mixed culture of common gut bacteria, including Escherichia coli, Bacteroides fragilis, and Lactobacillus acidophilus. Cultures were exposed to EMF at 900 MHz (similar to cell phone frequencies) for 24 hours, with and without the presence of 2 mM BHB.
Oxidative Stress Markers: We measured levels of malondialdehyde (MDA) and 8-hydroxy-2'-deoxyguanosine (8-OHdG) as markers of lipid peroxidation and DNA oxidation, respectively.
Gene Expression Analysis: RT-qPCR was used to assess changes in expression of genes related to antioxidant defenses, including superoxide dismutase (SOD) and catalase (CAT).
Microbiome Composition Analysis: 16S rRNA gene sequencing was performed to evaluate changes in gut microbiome composition under different experimental conditions.
In Vivo Ketosis Model: We used a mouse model to induce ketosis through a ketogenic diet. Fecal samples were collected and analyzed for microbiome composition and oxidative stress markers following EMF exposure. Blood samples were also collected to assess markers of oxidative stress and inflammation.
Results:
Oxidative Stress Markers: EMF exposure significantly increased levels of MDA and 8-OHdG in bacterial cultures (p < 0.001). The presence of BHB reduced these markers by 35% and 28%, respectively (p < 0.01).
Gene Expression: EMF exposure led to a 2.5-fold increase in SOD and CAT expression (p < 0.001). In the presence of BHB, this increase was further enhanced to 3.8-fold (p < 0.001).
Microbiome Composition: EMF exposure altered the relative abundance of certain bacterial species, with a 20% decrease in Lactobacillus spp. and a 15% increase in Enterobacteriaceae. BHB supplementation mitigated these changes, maintaining a composition more similar to the control group.
In Vivo Ketosis Model: Mice on a ketogenic diet showed increased resistance to EMF-induced changes in their gut microbiome composition and had lower levels of oxidative stress markers in fecal samples following EMF exposure compared to control diet mice (p < 0.01). Blood samples from ketogenic diet mice also showed reduced levels of oxidative stress markers and inflammatory cytokines compared to control diet mice (p < 0.05).
Discussion: Our results suggest that BHB may provide a protective effect against EMF-induced oxidative stress not only in gut bacteria but potentially in the bloodstream as well. The reduction in oxidative stress markers in the presence of BHB indicates its potential to mitigate EMF-induced damage. The enhanced expression of antioxidant genes in the presence of BHB suggests that ketones may boost the bacteria's innate defense mechanisms against oxidative stress.
The changes in microbiome composition observed with EMF exposure highlight the potential for environmental stressors to disrupt the gut ecosystem. The ability of BHB to maintain a more stable microbiome composition in the face of EMF exposure suggests that ketones may play a role in promoting microbiome resilience.
The in vivo results from our ketosis mouse model provide further support for the protective effect of ketones against EMF-induced changes in the gut microbiome and extend these findings to the bloodstream. The reduction in blood markers of oxidative stress and inflammation in ketogenic diet mice suggests that ketones may provide systemic protection against the harmful effects of EMF exposure.
The antioxidant and anti-inflammatory properties of ketones have been well-documented (Poff et al., 2015; Youm et al., 2015), and these effects could extend to the prevention of blood cell aggregation. In conditions of oxidative stress, red blood cells can undergo morphological changes and increased aggregation, leading to impaired blood flow and oxygen delivery (Gyawali et al., 2015). By reducing oxidative stress and inflammation, ketones may help to maintain normal blood cell function and prevent this harmful aggregation.
Furthermore, the gut microbiome and the bloodstream are not isolated systems. Gut bacteria and their metabolites can enter the bloodstream, influencing systemic inflammation and oxidative stress (Lau et al., 2017). By promoting a healthier, more resilient gut microbiome, ketones may indirectly contribute to a reduction in blood cell aggregation and associated health risks.
These potential systemic effects of ketones highlight the far-reaching implications of our findings. If ketones can indeed provide multi-level protection against EMF-induced oxidative stress, this could have profound implications for the prevention and management of a wide range of health conditions associated with oxidative stress and inflammation.
Conclusion: This study provides initial evidence supporting the hypothesis that ketones, particularly BHB, may offer protection against EMF-induced oxidative stress not only in the gut microbiome but potentially in the bloodstream as well. Our findings suggest that this protection may be mediated through direct antioxidant effects, enhancement of bacterial antioxidant defenses, promotion of a more resilient microbiome composition, and reduction of systemic oxidative stress and inflammation.
These results have broad implications for understanding the complex interplay between diet, the gut microbiome, and systemic health. They suggest that dietary interventions, particularly those that promote ketosis, could be a powerful tool for modulating the gut microbiome, enhancing its resilience to environmental stressors, and promoting overall health.
By reducing oxidative stress and inflammation, ketones may help to prevent harmful blood cell aggregation and maintain normal blood flow. This expands our understanding of the potential health benefits of ketones, suggesting that their protective effects may extend beyond the gut to the circulatory system.
Future research should investigate the specific effects of ketones on blood cell function and aggregation in the context of EMF exposure. Additionally, studies exploring the link between the gut microbiome, systemic inflammation, and blood cell health could provide further insights into the mechanisms underlying the protective effects of ketones.
In conclusion, this study opens up exciting new avenues for research into the health benefits of ketones and their potential to mitigate the harmful effects of EMF exposure. By providing multi-level protection against oxidative stress, ketones may represent a powerful tool for promoting overall health and well-being in an increasingly EMF-exposed world. These findings have the potential to inform the development of innovative strategies for preventing and managing a wide range of health conditions associated with oxidative stress and inflammation.
References: Clemente, J. C., Ursell, L. K., Parfrey, L. W., & Knight, R. (2012). The impact of the gut microbiota on human health: an integrative view. Cell, 148(6), 1258-1270.
Cryan, J. F., & Dinan, T. G. (2012). Mind-altering microorganisms: the impact of the gut microbiota on brain and behaviour. Nature reviews neuroscience, 13(10), 701-712.
Gyawali, P., Richards, R. S., Hughes, D. L., & Tinley, P. (2015). Erythrocyte aggregation and metabolic syndrome. Clinical hemorheology and microcirculation, 59(3), 257-276.
Kivrak, E. G., Yurt, K. K., Kaplan, A. A., Alkan, I., & Altun, G. (2017). Effects of electromagnetic fields exposure on the antioxidant defense system. Journal of microscopy and ultrastructure, 5(4), 167-176.
Lai, H., & Singh, N. P. (2004). Magnetic-field-induced DNA strand breaks in brain cells of the rat. Environmental health perspectives, 112(6), 687-694.
Lau, W. L., Kalantar-Zadeh, K., & Vaziri, N. D. (2017). The gut as a source of inflammation in chronic kidney disease. Nephron, 135(4), 256-263.
Lynch, S. V., & Pedersen, O. (2016). The human intestinal microbiome in health and disease. New England Journal of Medicine, 375(24), 2369-2379.
Newman, J. C., & Verdin, E. (2014). Ketone bodies as signaling metabolites. Trends in Endocrinology & Metabolism, 25(1), 42-52.
Poff, A. M., Ari, C., Arnold, P., Seyfried, T. N., & D'Agostino, D. P. (2015). Ketone supplementation decreases tumor cell viability and prolongs survival of mice with metastatic cancer. International journal of cancer, 135(7), 1711-1720.
Veech, R. L. (2014). Ketone ester effects on metabolism and transcription. Journal of lipid research, 55(10), 2004-2006.
Yakymenko, I., Tsybulin, O., Sidorik, E., Henshel, D., Kyrylenko, O., & Kyrylenko, S. (2016). Oxidative mechanisms of biological activity of low-intensity radiofrequency radiation. Electromagnetic biology and medicine, 35(2), 186-202.
Youm, Y. H., Nguyen, K. Y., Grant, R. W., Goldberg, E. L., Bodogai, M., Kim, D., ... & Dixit, V. D. (2015). The ketone metabolite β-hydroxybutyrate blocks NLRP3 inflammasome-mediated inflammatory disease. Nature medicine, 21(3), 263-269.
I'm so glad I ran into this study. I've been contemplating the potential benefits my diet has had in helping me withstand the harmful effects of extreme EMF exposure. I'm a T.I. and have discovered that the water main beneath my bed is emmiting 214+mG.
It causes all sorts of overall health and mental health issues. Extreme fluctuations of EMFs every night causes my heart rate and blood pressure to spike in conjunction, thus preventing me from sleeping. In addition, my kidneys and bladder begin to pulsate, resulting in sudden urgency to urinate multiple times a night. However, I've discovered that by simply placing my hand over my bladder, the pressure building up quickly subsides. Moreover, I experience cellular cascading all over my body.
Over time, I've grown concerned about the oxidative damage that is occurring as a result of sleep deprivation, dehydration, and activation of voltage-gated calcium ion channels, respectively. Despite these incidents being a regular occurrence, I'm surprised that I haven't become as debilitated as other individuals with similar experiences. This study confirmed hypothesis!
About 15 years ago, I developed an auto-immune issue which resulted in chronic urticaria and recurring fungal infections which led to lesions forming all over my body and scalp. Over the course of 4 years, I began studying nutrition and eventually refrained from eating processed sugars and carbohydrates. After seeing the results, I eventually switched to carnivore and have been healthier than I've ever been. Occasionally, I end up eating a meal with a lot of carbohydrates. I immediately feel the effects of exposure to high levels of EMFs begin to increase.
I'm sure you're already aware, our gut microbiome plays a large role in our immune systems. Maybe some time in the future, you can also put a study together on the effects of BHB on immunoassay screenings, fungal growth on the epidermis, and candida in the gut?
I wrote a research proposal last year regarding the potential role of gut microbiota on dopaminergic responses to identify the remaining levels of dopamine, postprandial glucose, in mice who lacked MCH neurons. Enterobacter in the digestive system produces a majority of serotonin, so it makes me wonder what other neurotransmitters our microbiota might play a role in producing and/or illiciting a response.
Looking forward to reading your future studies!